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Elucidation of the Inhibitory Effect of Phytochemicals with Kir6.2 Wild-Type and Mutant Models Associated in Type-1 Diabetes through Molecular Docking Approach |
Manaswini Jagadeb, V Badireenath Konkimalla, Surya Narayan Rath, Rohit Pritam Das |
Genomics Inform. 2014;12(4):283-288. Published online December 31, 2014 DOI: https://doi.org/10.5808/GI.2014.12.4.283 |
Elucidation of the Inhibitory Effect of Phytochemicals with Kir6.2 Wild-Type and Mutant Models Associated in Type-1 Diabetes through Molecular Docking Approach Inhibitory effect of acidic pH on OmpC porin: wild-type and mutant studies Retraction: Comparative molecular docking and molecular‐dynamic simulation of wild‐type‐ and mutant carboxylesterase with BTA‐hydrolase for enhanced binding to plastic Molecular docking studies of chromone derivatives against wild type and mutant strains of HIV-1 protease Molecular docking and QSAR studies for modeling the antimalarial activity of hybrids 4-anilinoquinoline-triazines derivatives with the wild-type and mutant receptor pf-DHFR Use of docking, cross docking and molecular dynamic simulations to rationalize the activity data of some S-dabos on wild-type and three mutant strains of reverse transcriptase Sustained miRNA-mediated Knockdown of Mutant AAT With Simultaneous Augmentation of Wild-type AAT Has Minimal Effect on Global Liver miRNA Profiles Wild-Type and Mutant B-RAF Activate C-RAF through Distinct Mechanisms Involving Heterodimerization Cotranslation of activated mutant p53 with wild type drives the wild-type p53 protein into the mutant conformation Understanding Molecular Mechanisms for Diabetes and Obesity through Mouse Models |